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Saturday, April 1, 2006

American multi-billionaire Donald Trump has announced plans to build a massive golf complex on an 800-acre site just north of Aberdeen, Scotland.

Speaking last night from the Trump Tower in New York, Trump said that the development will be akin to a “homecoming” for him. The ginger-haired property tycoon’s mother is from the Western Isles of Scotland.

The £300m project at Menie in Aberdeenshire will include both a links course and an inland course, and a 400-bedroom hotel as well as a golfing academy and holiday homes. The facilities are being built with the intention of attracting the Open Championship to the site, officially dubbed Trump International Golf Links, Scotland.

Fields and sand-dunes make up the landscape of the coastal site. Trump said of the location, “As soon as I saw it there was no question about it. I looked at 211 fantastic sites all over Europe, but here it is – the dune size and the ocean front. There is no piece of land that I have ever seen that is comparable to this.” The billionaire claims that the complex, set to be his only development outside the United States, will be the “best course in the world.”

Local and national political figures, including Scottish First Minister Jack McConnell, have welcomed the announcement with open arms, as the project is expected to bring in an additional £157m to the region’s economy within the first ten years. Scottish tourism company VisitScotland is also optimistic, expecting an influx of international visitors to play on (or simply visit) the site. Plans to expand the A90 road running past the site in the near future will enable more road traffic to and around the area. An £60m expansion of Aberdeen Airport will also allow for vastly increased international air links, akin to those operated using Boeing 767 aircraft.

Work on the golf complex itself is set to begin in September this year, with the Trump Organisation hopeful for a Spring 2008 opening date.

Tuesday, January 13, 2015

A Sheriff in Aberdeen yesterday jailed gamekeeper George Mutch for crimes against birds of prey. It is believed to be the first time anybody has been imprisoned for raptor persecution in Scotland.

Mutch, 48, killed a protected goshawk, and used illegal traps to capture a buzzard and a second goshawk. He was convicted last month of four offences, which he committed in 2012 in Aberdeenshire.

The Royal Society for the Protection of Birds (RSPB) used hidden cameras to record Mutch’s crimes, one of the first times such footage has featured in a Scottish wildlife trial. The video shows Mutch remove a juvenile goshawk from one trap and beat it to death with a stick. He removes the other goshawk and the buzzard, putting them in sacks which he then walks away with. Their fate is unknown.

Sheriff Noel McPartlin called Mutch’s conduct “a determined course of action. Having regard to the gravity of the offence [of killing the goshawk], I am of the view that there is no other method of dealing with you which is appropriate to this case other than the imposition of a custodial sentence.” Sara Shaw, Scotland’s procurator fiscal (prosecutor) for wildlife crime, explained “Goshawks in particular are rare birds: the court heard evidence in this case that there are only about 150 nesting pairs in Scotland.” She said the law was there “to preserve Scotland’s natural heritage, including the wildlife that forms part of it.”

David McKie, defending, detailed other sanctions his client had already suffered. His employers on the Kildrummy Estate have suspended him, and police have seized his guns; he may lose his gun licence because of his conviction. The Scottish Gamekeepers Association separately announced Mutch’s expulsion from the group’s ranks.

RSPB Scotland’s Duncan Orr-Ewing called the sentence a “historic, landmark result” and “a turning point, sending a clear message to those determined to flout our laws”. Landowners’ body Scottish Land and Estates also had no sympathy: “The illegal killing of any bird of prey is unacceptable and anyone who engages in such activity can, rightly, expect to feel the full weight of the law.”

The RSPB said it may be the first such jailing anywhere in the UK. The group said other wildlife crimes, such as against badgers, had previously attracted prison terms. “We would like to thank the Crown Office and Police Scotland for helping to bring this case to a successful conviction, as well as the exemplary work of the RSPB Scotland investigations team” said Orr-Ewing. He added “Wildlife criminals must expect no sympathy from now on.”

Scotland’s goshawk is the northern goshawk, Accipiter gentilis. Internationally, the species has a wide range and a large population through much of Europe, North America, Russia, and northern Asia. It is therefore ranked as least concern by the International Union for the Conservation of Nature’s Red List of Threatened Species. However, the bird does face persecution and nest robbing by humans. Indirect human impacts include deforestation and, according to a 2012 study, wind farm developments. Scotland has invested heavily in wind power over the last decade.

Thursday, December 13, 2007

Victoria Wyndham was one of the most seasoned and accomplished actresses in daytime soap opera television. She played Rachel Cory, the maven of Another World‘s fictional town, Bay City, from 1972 to 1999 when the show went off the air. Wyndham talks about how she was seen as the anchor of a show, and the political infighting to keep it on the air as NBC wanted to wrest control of the long-running soap from Procter & Gamble. Wyndham fought to keep it on the air, but eventually succumbed to the inevitable. She discusses life on the soap opera, and the seven years she spent wandering “in the woods” of Los Angeles seeking direction, now divorced from a character who had come to define her professional career. Happy, healthy and with a family she is proud of, Wyndham has found life after the death of Another World in painting and animals. Below is David Shankbone’s interview with the soap diva.

Tuesday, July 12, 2005

Dramatic developments today have revealed new information about last Thursday’s bomb attacks on London. One person has been arrested after a series of raids in Leeds.

The four men alleged to be responsible for the bombings travelled by train from Leeds to King’s Cross on Thursday morning, where they were seen on CCTV at 8.30am. Three of them, likely British citizens of Pakistani origin, came from Leeds while another joined them en route. One of the three from Yorkshire was reported missing by his family at 10pm that evening.

Three of them then boarded underground trains. The fourth alleged bomber – the man reported missing from Leeds – died onboard the bus, but there are no clear reasons for why he did not follow the same pattern as the others. It has been confirmed that at least three of the four bombers died in the blasts. Forensic evidence and items belonging to all of the men were found in the debris of the target vehicles.

Police also found a car at Luton train station, which is on the route from Leeds to King’s Cross. A series of controlled explosions was carried out on the car. It is understood that one of the bombers drove equipment in the car to Luton, while the others travelled by train.

Materials used to make explosives were also found at one of the houses in Leeds.

A second car was found on the property of a recovery company in Leighton Buzzard; it had been ‘routinely recovered’ from Luton train station on the evening of Tuesday, July 12. Bedfordshire Police, working in conjunction with the Metropolitan Police and the British Transport Police, sealed off the area within a 50 metre radius of the vehicle. The area was re-opened at 9.30pm BST, although a police presence remained until the next day. It has not yet been revealed why the car was towed away or what was contained inside it.

Saturday, March 10, 2012

The Kenyan government has dismissed 25,000 striking health workers, mostly nurses, citing failure to heed government orders to recommence work and concern for the welfare of hospital patients. Speaking on behalf of the government, Alfred Mutua stated the workers were dismissed “illegally striking” and “[defying] the directive … to report back to work”, which he called “unethical”. The government asks that “[a]ll qualified health professionals, who are unemployed and/or retired have been advised to report to their nearest health facility for interviews and deployment”, Mutua stated.

The workers, who had been on strike for four days, were wishing to have improvements made to their wages, working conditions, and allowances. The strikes have caused a significant number of Kenyan hospitals to cease operations. According to Kenya Health Professionals Society spokesperson Alex Orina, the average monthly wage plus allowances for health workers in Kenya is KSh25,000 (£193, US$302 or €230) approximately. With an increasing number of reports of patients neglected in hospitals emerging, two trade unions met with the Kenyan government yesterday and negotitated a return to work, although a significant proportion of demonstrators defied the agreement, The Guardian reported.

Orina told Reuters the dismissals were “cat-and-mouse games, you cannot sack an entire workforce. It is a ploy to get us to rush back to work, but our strike continues until our demands are met”. Frederick Omiah, a member of the same society, believed the government’s actions would “make an already delicate and volatile situation worse”, expressing concern that demonstrations may continue in the capital Nairobi, amongst other locations. Kenya Medical Practitioners, Pharmacists and Dentists Union chairperson Dr. Victor Ng’ani described government actions as “reckless”.

Mutua said the health workers were “no longer employees of the government” and had been eliminated from the payroll. While Ng’ani told the BBC of difficulties with finding other workers as skilled and experienced, Mutua reportedly stated that this would not be an issue. “We have over 100,000 to 200,000 health professionals looking for work today,” Mutua commented. “There will be a lag of a day or two … but it is better than letting people die on the floor, at the gate, or suffer in pain”.

Sunday, June 20, 2010

Nicolas Sarkozy visited the UK today to celebrate the 70th anniversary since Charles de Gaulle made his war broadcast. The French president and the Prince of Wales laid wreaths at the statue of Charles de Gaulle in London.

Earlier, Mr Sarkozy and his wife Carla Bruni visited the BBC radio studio where the famous broadcast was made on BBC France. The broadcast at the time was said to have been listened to by a very small number of listeners. Carla Bruni and Nicolas Sarkozy unveiled a plaque and viewed a tapestry given to the BBC by France as a note of gratitude after World War II.

Nicolas Sarkozy and British PM David Cameron met with 200 veterans during a ceremony at the Royal Hospital Chelsea. Nicolas Sarkozy awarded the Légion d’honneur to six World War II veterans, three of whom were British.

In a short speech, Mr Cameron said the anniversary was a “reminder that Britain and France are not just neighbours in the geographical sense but also in the emotional sense.” Mr Cameron held an hour of talks with the French president at Downing Street primarily focusing on economics, Afghanistan, and other foreign policy matters. The visit is the first by a French president to mark Gen de Gaulle’s broadcast on June 18, 1940. In the stirring radio appeal, Gen de Gaulle declared himself leader of the “Free French”, spawning the French Resistance, which went on to play a crucial role in defeating the Germans.

Mr Sarkozy’s last official visit to the UK was in March 2008, when he was also accompanied by his wife.

Saturday, October 1, 2011

Last week, Nature Genetics carried twin studies into the genetics of bipolar disorder and schizophrenia. This special report examines the month’s research into the illnesses in detail, with Wikinews obtaining comment from experts based in Australia, the United States, and the United Kingdom ahead of the U.S. Mental Illness Awareness Week, which starts tomorrow.

Eleven genetic regions were identified; seven of these were for schizophrenia and five of those were hitherto undiscovered. The parallel studies, conducted separately, examined more than 50,000 people worldwide and identified two genetic loci associated with both diseases.

Little is known about the two illnesses, each of which affects around 1% of people and is treated with strong medication. Bipolar sufferers experience extremes of mood – depression and mania, hence the previous name “manic depression” for the illness. Schizophrenia is associated with hearing voices, chaotic thoughts, and paranoia. There is no known cure.

The latest research examined both the healthy and the afflicted, using computers to scan genomes. Inheritance was thought to be a factor from prior knowledge of the diseases as a familial trait, but the original desire had been to isolate a single faulty gene. Instead it has become apparent that the genetic factors are many; in the case of schizophrenia, at most around 30% of the genetic components are thought to have been identified.

The University of Chicago’s Pablo Gejman, a lead researcher on the schizophrenia study, explained to Wikinews in a telephone interview from Buenos Aires, Argentina that “One of the goals of genetic research is to find druggable targets” – to “find treatments at the root of the problem”.

Whilst noting that there is no guarantee the genetic code identified is druggable, Gejman named calcium-activated neurochemical channels in the brain as candidates for new drugs. The channels were linked to schizophrenia in the study.

Gejman explained that a genetic locus called mir137 “suggests an abnormality of gene regulation.” The diseases are so poorly understood that it is uncertain if they are in fact two components of a single spectrum, or even each comprised of multiple illnesses.

The new and “provocative data” gathered showed the significant loci identified were “not part of the pre-existent hypothesis.” Calling this “interesting”, Gejman added that the team found no evidence that dopamine receptors are involved; current drug treatments target dopamine receptors. The findings are “not related to anything we thought we knew [about schizophrenia],” he told our correspondent.

Quizzed about the possibility variations in the genetic factors involved in expressing the diseases explained the variation seen in symptoms, Gejman was uncertain. “We will have the answer, probably, only when we sequence the whole [human] genome.” He notes that the relationship between genotype and phenotype is unclear, and that “We know very little of the genetic architecture of schizophrenia and” other disorders.At the time the results were published, participating scientist Professor Rodney Scott from the University of Newcastle in Australia said “The strength of this research is in the numbers. The findings are robust and give us a lot of statistical power to identify the genetic determinants of schizophrenia.” Scott told Wikinews that “If any single centre tried to undertake such a study, it would require millions of pounds. Since it was a collection of data from across the world the costs were spread. In this era of financial difficulty it will become increasingly difficult to secure funding for this type of project even though the pay-offs will be significant.”

Gejman expressed similar sentiment. “The research budget is not growing, which makes [funding] difficult,” he said, though he felt the cost “is not prohibitive because of the benefits.” “I think that it was money well invested” and “very well spent for the future,” he said, adding that organisations in Europe and the US were aware of the importance of such research.

Gejman also agreed on reliability – the study is “Very reliable because of the sample size; that should provide robust results… [we] have worked with a much larger sample than before.” Scott told us it was “a highly reliable study” that has the potential to lead to new treatments “in the long run”.

Another point was the two genetic loci identified as common to both – how much support do they lend to the notion the diseases are linked? “Until more information is available it is really only suggestive,” says Scott. “Strong enough to say there may be potentially a common pathway that bifurcates to give rise to two diseases.”

“It is an excellent demonstration,” said Gejman “because you have the same chains that are common to both disorders, in fact not just the same chains but also the same alleles.” He stressed uncertainty in how strong the relationship was, however.

Scott said examining how the variation of genetic factors may translate into varied symptoms being expressed “certainly is a good target for future research”; “It is not known how many genetic factors contribute to either of these diseases but it is likely that not all are necessary to trigger disease.” “New questions will always arise from any major study,” he told our reporter. “Certainly, new questions about bipolar and schizophrenia are now able to be formulated on the basis of the results presented in the two reports.”

These weren’t the only studies to look at the two diseases together in September. The British Medical Journal carried research by a team from the University of Oxford and King’s College London that examined mortality rates in England for schizophrenia and bipolar sufferers. They found both groups continued to suffer higher mortality rates than the general population – whilst these included suicides, three quarters of deaths were down to ailments such a s heart conditions. General death rates dropped from 1999 to 2006, but sufferers below 65 saw their death rate remain stable – and the over-65 saw theirs increase.

“By 2006, the excess risk in these groups had risk to twice the rate of the general population, whereas prior to that it had only been 1.6 times the risk, so it increased by almost 40%,” said Dr Uy Hoang of Oxford. The study looked at every discharged inpatient with a diagnosis of either condition in England in the relevant time.

Hoang said at the time of the research’s release that doctors should devote attention to predicting and preventing physical illness associated with mental disorders. His study comes at a time when the UK has launched a “no health without mental health” strategy which does attempt to screen for physical illnesses coinciding with mental illnesses. The government aims to reduce the death rate of those with mental disorders.

Rodney Scott described this research result to Wikinews as “Possibly” connected to genetic association with other hereditary ailments, such as cardiovascular disease; he told us another possibility is that “The continued raised mortality rates may be associated with the diseases themselves.”

“We believe the NHS [National Health Service] and Department of Health need to do more to support research and service development for people with bipolar disorder,” Wikinews was told by Suzanne Hudson, Chief Executive of London-based British charity MDF The Bipolar Organisation. “The provision of specialist services for bipolar is very limited in the UK and the demand for our services is unprecedented.”

“A genetic test for bipolar would be a useful tool but the science and ethics are very complex,” Hudson told us, referring to the Nature Genetics genetic study. “Just because someone has ‘bipolar genes’ does not mean they might go on to develop it. Family studies of bipolar show that this is a likely outcome of genetics research in this area. Even if it were possible to accurately predict bipolar in this way, questions about how you treat that person are difficult. For example do you start medication that is not necessary at that point in time?”

“Current treatment is not satisfactory” because it does not always work and has “side effects,” Gejman told us. Robert Whitaker, a US medical journalist and book author, told an audience in New Zealand at the end of August that evidence suggests antidepressant drugs may make children and teenagers worse – “You see many become worse and end up with a more severe diagnosis, like bipolar illness,” and the suicide risk may increase.

Whitaker blames commercial interests. “The adult market appeared saturated, and so they began eying children and teenagers. Prior to this, few children and youth were seen as suffering from major depression, and so few were prescribed anti-depressants.”

One possible alternative, raised by a connection between depressive illness and inflammation, is aspirin and similar compounds. “The link between inflammation and mood disorders has been known for sometime and the use of aspirin and other drugs in depression is now becoming more common in the literature,” Hudson says. “Any new treatments for bipolar, which is a very complex and co-morbid illness, has to be a good thing.”

Professor Dr. Michael Berk, chairman of psychiatry at Australia’s Deakin University, recently gave a talk to just this effect. Speaking at this year’s Congress of the European College of Neuropsychopharmacology, held this past month, he also highlighted statins as a treatment. Recognising the link to physical ailments, he told an interviewer “The brain does not exist in isolation, and we need to understand that pathways similar to those that underpin risks for cardiovascular disorders, stroke, and osteoporosis might also underpin the risk for psychiatric disorders, and that other treatments might be helpful.”

Berk also touched upon speed of diagnosis and treatment; “Early interventions can potentially improve the outcome” of bipolar sufferers, he told his audience. MDF The Bipolar Organisation claim an average of ten years is possible before a person is diagnosed. “This clearly is an issue, if we believe that earlier diagnosis and treatment facilitate better outcomes,” Berk told Wikinews. Though he questions the effectiveness of currently-used drugs on advanced bipolar cases, he does not go so far as to say drugs are actively harmful. He told us “it appears that our best treatments work best earlier in the illness course; and that seems to apply to psychotherapy and pharmacotherapy.”

Berk has already performed research using statins which suggests they can form a treatment. He now seeks funding for research involving aspirin. On funding, he tells Wikinews “psychiatric disorders comprise between 16% and 22% of the burden of disability (depending on who measures it), attracts^[sic] just over 6% of the clinical budget at least in Australia and 3% of the research budget. Research as a discretionary spending item is at great risk.”

Berk’s research, in the past, has been funded by companies including GlaxoSmithKline. Hudson told Wikinews this did not concern her charity; in fact, they welcomed it. “We believe it is important pharmaceutical companies continue to invest in the development of new medications for bipolar. This is how it works in all other health specialities and mental health should be no different.”

“There is a need for greater education for mental health professionals and GPs [general practitioners] about bipolar [in the UK],” she told us. “As the national bipolar charity we receive many, many calls and requests from GPs and other health professionals for our leaflets and information sheets which is fantastic. We very much welcome opportunities to work together for the benefit of individuals affected by bipolar.”

Wikinews contacted the UK’s National Institute for Clinical Excellence (NICE) to discuss issues raised in this article, including future treatments, genetic screening, and mortality rates. NICE did not respond.

Might statins and/or aspirin improve treatment – might they be cheaper, perhaps, or safer? “This is an area of research promise,” says Berk, “however it is too early to make any clinical treatment claims; [all] we can say is that this needs to be studied in properly designed trials capable of giving a more definitive answer.” And what of possible explanations for the increased mortality rate observed in England? Should researchers look at whether bipolar influences more than just the brain, or if it is linked to other genetic conditions?

“For sure,” he told us. “There is new evidence that similar pathways contribute to the risk for both medical and psychiatric illness, both in terms of lifestyle factors, and biomarkers of risk.”